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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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Excessive glutathione intake contributes to chemotherapy resistance in breast cancer: a propensity score matching analysis World Journal of Surgical Oncology Springer Nature Link International Journal of Oncology Glutathione in Cancer Cell Death Glutathione Dependent Pathways in Cancer Cells Glutathione and Xanthine Metabolic Changes in Tamoxifen Resistant Breast Cancer Cell Lines are Mediated by Down Regulation of GSS and XDH and Correlated to Poor Prognosis Association of glutathione S transferase T1, M1, and P1 polymorphisms TCRM Dove Medical Press
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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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